“Huntington’s disease presents an ideal vantage point to study neurodegenerative disease, because we know the misfolded protein that’s responsible,” says Martin Duennwald, formerly a postdoctoral researcher in the lab of Whitehead Member Susan Lindquist. “But we don’t understand how this protein causes cellular damage and death for the neurons that are affected.” In a study
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When bacterial infections cause us to get really sick, the only way to get better is to go to the doctor. With the help of antibiotics, we can start to recover quickly. One such antibiotic is Augmentin which belongs to the penicillin family of drugs.
A bacterial infection is different to a viral infection, therefore the treatment is also different. Many bacteria live in our bodies – these are good bacteria because they help us to stay healthy. There are millions of bacteria everywhere but only a tiny percentage of them can make us ill.
When the harmful ones enter our bodies they often upset the balance in our system by destroying the good bacteria. Our bodies try hard to fight them off and our immune systems get charged up to try and kill them. When the immune system is firing on all cylinders, we start to feel terribly sick and break out in a high fever and get aches and pains everywhere.
If the immune system cannot kill the harmful bacteria, it will need some help from an antibiotic like Augmentin. Once this medication gets into the system, it immediately starts to help the immune system fight the bacteria and we start to get better. To help this process we need to rest, that’s why the doctors usually tell us to stay in bed for a few days.
Augmentin is ideal for many types of bacterial infections – some common ones being ear infections, sinusitis, bronchitis, pneumonia, urinary tract infections, and certain skin infections. It is important to point out that this drug will not help with colds or influenza – these are viral infections and will need a completely different type of medication.
Most times we are able to fight infections on our own. But if we are slightly run down the bad bacteria are able to multiply rapidly. This is when we need the help of an antibiotic like Augmentin.
In the same way that winter is commonly known to be the “flu season,” a new study suggests that the dog days of summer may well be the “bacterial infection” season. Researchers have discovered that serious infections caused by gram-negative bacteria can go up as much as 17 percent with every 10 degree increase in
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We are always being told by marketers of healthy yogurts that the human gut contains a bustling community of different bacteria, both good and bad, and that this balance is vital to keeping you healthy. But if you target the disease-causing bacteria with medicine, what might be the collateral damage to their health-associated cousins that
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Scientists have identified the structure of a key component of the bacteria behind such diseases as whooping cough, peptic stomach ulcers and Legionnaires’ disease. The research, funded by the Wellcome Trust and the Biotechnology and Biological Sciences Research Council (BBSRC), sheds light on how antibiotic resistance genes spread from one bacterium to another. The research
Full Post: Discovery of novel ways to halt the spread of antibiotic resistance
Like firemen fighting fire with fire, researchers at the University of Illinois and the University of Massachusetts at Amherst have found a way to fool a bacteria’s evolutionary machinery into programming its own death. “The basic idea is for an antimicrobial to target something in a bacteria that, in order to gain immunity, would require
Full Post: Scientists fool bacteria into programming own death
It’s as simple as A, T, G, C. Northwestern University scientists have exploited the Watson-Crick base pairing of DNA to provide a defensive tool that could be used to fight the spread of antibiotic resistance in bacteria — one of the world’s most pressing public health problems. The resistant nasty pathogens cause thousands of deaths
Full Post: Blocking mechanism found for antibiotic resistance in bacteria